Nutrition for gynecological diseases

Candidiasis is a yeast disease caused by the Candida albicans strain. The first symptoms of the development of thrush may be itching of the mucous membrane, burning, the appearance of white discharge similar to cottage cheese, and irritation. Then inflammation and dryness appear. This disease is not dangerous, but it causes a lot of discomfort to a person for a long time. A diet for thrush will help not only cope with the symptoms, but also forget about this problem forever.

What is candidiasis

Candidiasis is caused by yeasts of the genus Candida.
These microorganisms are classified as opportunistic. They are detected on the mucous membranes of the intestines and genitals. If the immune system is strong, it can control the number of fungi, but when it decreases, microorganisms begin to multiply uncontrollably, causing inflammation of the mucous membrane and other manifestations of the disease. The following unfavorable factors contribute to the development of candidiasis:

• insufficient hygiene;
• dysbacteriosis;
• alcohol abuse;
• improper installation of catheters;
• taking antibiotics;
• diabetes;
• a sharp decrease in immunity;
• avitaminosis;
• stress, etc.

Alcohol contributes to the development of candidiasis.
In addition, consuming foods rich in refined carbohydrates and sugar increases the risk of developing this fungal disease. Symptoms of candidiasis depend on which organ is affected.

Manifestations of the most common urogenital type of disease include:

• itching;
• redness of the mucous membrane;
• curdled discharge;
• discomfort during sexual intercourse;
• unpleasant odor;
• burning.

When the oral area is affected, a white coating appears on the tongue. In addition, the formation of cracks and gray plaques is possible. The mucous membrane turns red and swells.

Paradoxically, this is partly due to the successes that modern medicine has achieved in the treatment of cancer and in the fight against pathogens of serious infections. In addition, it is obvious that in recent decades there has been an increase in the intensity of the impact on the human body of external factors that cause the development of immunosuppressive conditions. In addition, the end of the 20th century was marked by a significant spread of a fatal disease - HIV infection. Pathological conditions caused by the influence of fungi of the genus Candida on the human body also belong to the category of opportunistic mycoses. Characteristics of the pathogen Representatives of the genus Candida are yeast fungi related to Ascomycetes. The genus Candida includes various species (Candida spp.), among which the main ones as causative agents of candidiasis are: Candida albicans, Candida tropicalis, Candida parapsilosis, Candida glabrata, Candida dubliniensis, Candida lusitaniae, Candida krusei. Candida spp. They are a component of microflora that is symbiont for humans. In 10–25% of the population without clinical signs of fungal infection, Candida spp. are inconsistently detected in the oral cavity, in 65–80% - in the intestinal contents. Candida spp. are found in the detritus of gastroduodenal ulcers in approximately 17% of cases. According to modern ideas, under normal conditions the main “habitat” of Candida spp. in the human body is the intestines. The proportion of these fungi in the intestinal microbial population is negligible. Growth of colonies of Candida spp. in the human body it is partly regulated due to the work of the immune system. The main place in this process is occupied by the links of nonspecific immunity - mononuclear phagocytes (monocytes/macrophages) and polymorphonuclear leukocytes [1,3]. Bacteria - intestinal symbionts - also play a very important role in limiting the growth of the Candida population. Normal microflora inhabiting the lumen of the gastrointestinal tract produces substances with antibacterial activity (in particular, bacteriokines and short-chain fatty acids), which prevent the introduction of pathogenic microorganisms and excessive growth, the development of opportunistic flora. Escherichia coli, enterococci, bifidobacteria and lactobacilli have the most pronounced antagonistic properties. The term “candidiasis” implies a pathological process, the basis of which is the excessive growth of Candida primarily in the gastrointestinal tract and secondarily in other areas (on the mucous membrane of the genitals, bronchi, in parenchymal organs) [1,3,5]. Virulence factors of Candida Fungi of the genus Candida are adhesive to epithelial cells. Attachment to the mucous membrane is one of the conditions for further invasion of the microorganism into the underlying tissue. The adhesion capacity of different Candida species varies considerably; this ability is highest in C. albicans, C. tropicalis, C. dubliniensis; the smallest is in Candida glabrata, Candida krusei. Mucin, a glycoprotein of the cell wall of epithelial cells, plays an important role as a protective barrier that prevents the microbe from attaching to the mucous membrane. Factors of aggression of these microorganisms include proteases and glycosidases that can intensively break down mucin. The ability to rapidly form filaments of pseudomycelium is also considered as a virulence factor; this feature is most pronounced in C. albicans. The virulence of microorganisms is subject to intraspecific variability, depending on the genotype [1,3]. Risk factors for the development of candidiasis As general factors predisposing to the development of candidiasis of the digestive organs and subsequent lympho-hematogenous dissemination of fungi, as in the cases of other opportunistic infections, are conditions in which there is a lack of immune defense of the body. They include: 1) Physiological immunodeficiencies (newborn period and early childhood, senile immunodeficiency, pregnancy, stress conditions). 2) Congenital immunodeficiencies (Di-George, Nezelof, Shediac-Higashi syndrome, etc.). 3) Infection with the human immunodeficiency virus in the terminal stage (acquired immunodeficiency syndrome). Mycoses make up 70% of the clinical manifestations of HIV infection. 4) Oncological diseases (including hematological malignancies); chemotherapy of malignant tumors, accompanied by severe granulocytopenia (<1x109 granulocytes per l). The development of candidiasis during antitumor therapy is usually associated with suppression of the functions of the immune system, observed as an undesirable effect of treatment. At the same time, there are suggestions that cytostatics and radiation therapy inhibit the protective functions of the intestinal epithelium and promote the translocation of Candida to other organs. During chemotherapy, the percentage of fungal microorganisms adhering to enterocytes increases. 5) Autoimmune and allergic diseases, especially when prescribing glucocorticosteroids. 6) Organ transplantation (use of immunosuppressants in the post-transplant period). More than half of patients scheduled for bone marrow transplantation have a picture of systemic candidiasis even before transplantation. 7) Endocrinopathies (decompensated diabetes mellitus, autoimmune polyglandular syndrome). Other diseases and conditions (shock conditions, anemia, hepatitis, cirrhosis of the liver, chronic infections, malabsorption syndrome, etc.). In pathological conditions accompanied by a decrease in blood supply to the intestine, the translocation of fungi into other tissues increases. Digestion and absorption disorders are accompanied by excessive growth of microorganisms in the intestinal lumen. 9) Antibiotic therapy (usually long-term use of broad-spectrum drugs). The introduction of antibacterial drugs can lead to an imbalance in the microbial biocenosis of the intestine, as a result of which excessive development of the candidal population in the intestine is possible. Prescription of tuberculostatic drugs for 3–4 months. accompanied by the development of intestinal candidiasis in 58–62% of cases. The risk of developing idiopathic antibiotic-associated diarrhea depends on the dose of antibiotic administered; this disease usually occurs without fever and leukocytosis in the blood and in the absence of laboratory signs of Clostridium difficile infection. It is believed that in about a third of cases, the development of idiopathic antibiotic-associated diarrhea is caused by intestinal candidiasis. 10) Unbalanced nutrition (deficiency in the diet of proteins, vitamins). Animal experiments have shown that insufficient protein intake into the body is accompanied by a decrease in the phagocytic and bactericidal activity of the macrophage component, incomplete phagocytosis and an increase in the permeability of the intestinal barrier to Candida albicans [1,3,4,5]. According to autopsy data, candidiasis of the duodenum, small and large intestine is found in approximately 3% of dead patients (only macroscopic changes were taken into account). Moreover, half of the cases occur in patients receiving chemotherapy for malignant neoplasms [1]. Pathogenesis of the development of intestinal candidiasis Research in recent years has largely clarified the patterns of interaction of Candida fungi with the host organism. According to modern concepts, two fundamentally different mechanisms of the pathogenesis of candidiasis of the digestive organs can be distinguished: invasive and non-invasive candidiasis [3]. Invasive candidiasis is caused by the introduction of a filamentous form of the Candida fungus into tissues. The first stage of candidiasis, as an infectious process, is adhesion to epithelial cells, then invasion occurs into the epithelial layer, penetration beyond the basement membrane. These manifestations of microbial aggression macroscopically correspond to erosive and ulcerative defects of the intestinal wall of various sizes and shapes, cracks, membrane deposits (similar to those in pseudomembranous colitis), polypoid or segmental circular formations. With progressive invasion, the development of lympho-hematogenous dissemination of fungi is possible (systemic candidiasis with damage to the mucous membranes of other organs; generalized candidiasis with damage to visceral organs). An experimental model of animals orally infected with C. albicans under conditions of immunosuppression showed the initial occurrence of erosions and ulcers of the mucous membrane in the ileum, colonization of intestinal lymphoid formations, their necrosis and further dissemination. The absence of macroscopic changes should probably not be considered as evidence against invasive candidiasis. In disseminated forms of candidiasis, multinucleated giant cells containing fungi are found in the lymph nodes and ducts, which probably reflects the phenomenon of incomplete phagocytosis. Invasive candidiasis is more often observed in organs lined with stratified squamous epithelium (oral cavity, esophagus), and less often with columnar epithelium (stomach, intestines), which is probably due to the peculiarities of local immune defense. Non-invasive candidiasis is not accompanied by the transformation of the fungus into a filamentous form; There is an excessive growth of its colonies in the lumen of a hollow organ - the intestine. It is assumed that the disruption of cavity and parietal digestion, the penetration of microbial components and metabolites into the systemic bloodstream, and the development of a more or less pronounced systemic immune-inflammatory reaction are of great pathogenetic importance. Based on all of the above, it should be emphasized once again that mucosal candidiasis of extraintestinal localization or generalized candidiasis with damage to parenchymal organs is a manifestation of the translocation of fungi from the intestinal lumen, where the bulk of these saprophytic fungi are represented. Candidiasis of extraintestinal localization (for example, the oral cavity or genitals) is a manifestation of systemic candidiasis, “originating” from the intestines. The following is a classification of candidiasis of the digestive organs: 1. Oro-pharyngeal candidiasis (cheilitis, gingivitis, seizures, glossitis, stomatitis, pharyngitis). 2. Candidiasis of the esophagus (complications – bleeding, stricture). 3. Gastric candidiasis: – diffuse (specific erosive-fibrinous gastritis); – focal (secondary to gastric ulcer). 4. Intestinal candidiasis: – invasive diffuse; – focal (secondary for duodenal ulcer, with nonspecific ulcerative colitis); – non-invasive (overgrowth of Candida in the intestinal lumen). 5. Anorectal candidiasis: – invasive candidiasis of the rectum, – perianal candidal dermatitis. Below we describe the main manifestations of candidiasis of the intestine, which, as mentioned above, is the basis for the development of systemic manifestations. Clinical picture of intestinal candidiasis Features of the course of intestinal candidiasis are not clearly defined, not well studied and little known to most practicing doctors. It is characteristic that in patients who died from various causes and whose autopsy revealed macroscopic changes in the intestine corresponding to invasive candidiasis, during their lifetime, as a rule, very scanty symptoms from the gastrointestinal tract were noted, and the endoscopic diagnosis was often erroneous. Often, when single ulcerations of the intestinal wall are detected, the doctor finds it difficult to interpret them, and based on the results of a morphological study, a conclusion is given about nonspecific inflammatory changes in the edges of ulcerative defects, while a targeted mycological study is not carried out. Clinical manifestations of intestinal candidiasis may vary depending on the level of damage. With diffuse invasive intestinal candidiasis, there are manifestations of enterocolitis: complaints of spastic abdominal pain, flatulence, the presence of pathological impurities in the stool (blood and mucus), there are usually signs of systemic candidiasis (damage to the mucous membranes of the oral cavity, genitals). Endoscopic examination reveals changes similar to fibrinous-ulcerative colitis. With invasive focal intestinal candidiasis, the manifestations of the disease may resemble a persistent, resistant to traditional therapy, course of duodenal ulcer or nonspecific ulcerative colitis. With non-invasive intestinal candidiasis, patients complain of loose stools, flatulence, abdominal discomfort, with positive clinical and laboratory dynamics when treated with antifungal drugs. With invasive candidiasis of the rectum, symptoms of proctitis (pain, tenesmus, pathological impurities in the stool) may be observed. In some cases, this is accompanied by the phenomenon of perianal candidodermatitis. Intestinal candidiasis is often accompanied by low-grade fever [1,3,5]. Complications As complications of intestinal candidiasis, the development of intestinal perforation, penetration of ulcers into surrounding organs, bleeding, generalization with damage to parenchymal organs, and the development of fungal sepsis are possible. Damage to parenchymal organs (liver, gallbladder, pancreas, etc.) very often accompanies deep neutropenia (less than 500 neutrophils per mm3 of blood) and is observed in the terminal phase of AIDS [1,3,5]. The mortality rate for invasive candidiasis reaches 25–55%. For some categories of patients (transplant recipients, patients with acute leukemia), invasive mycoses are the main cause of death. Diagnosis The question of diagnosing intestinal candidiasis and determining indications for antifungal therapy is extremely important. In recognizing mucosal candidiasis, it is necessary to distinguish between physiological “candidiasis” and the infectious process caused by this fungus. For the diagnosis of candidiasis, the detection of Candida in sterile fluids (cerebrospinal fluid, lavage fluid, peritoneal fluid, etc.) or the detection of fungi in tissues (changes such as granulomas with necrosis are often found) are absolutely informative. To increase the sensitivity of cultural and morphological methods for diagnosing candidiasis, it is recommended to examine several biopsies of the mucous membrane. The biopsied tissue is collected in 2 sterile Petri dishes or sterile jars with a screw cap; one sample is filled with a 10% formaldehyde solution and sent for histological examination, the second is used for mycological examination. The material is transported to the microbiological laboratory, protected from direct sunlight. It is necessary that the material be delivered for mycological examination no later than 1 hour after collection when stored at room temperature or no more than 3 hours when stored at +4°C. Microscopic examination must be carried out in native and stained preparations. The CHIC reaction (treatment with chromic acid) or its modification - Gridley staining - allows you to identify the pathogen in a tissue or smear by staining the polysaccharide components of the cell wall; To suppress the color of surrounding tissues, a “counter-paint” with light green, methanil yellow, etc. is used. In this case, only invading fungal cells are detected, while at the same time it is impossible to judge the reaction of the surrounding tissues. Therefore, it is also necessary to evaluate preparations “counter-stained” with hematoxylin and eosin. Pseudomycelium of Candida can also be detected in an impression smear of the mucous membrane or an impression smear from the bottom of the ulcer (Romanovsky-Giemsa stain). An imprint is made on the surface of a dense nutrient medium in a Petri dish with a piece of tissue being examined, then sieving is carried out with a loop. The same piece of tissue is placed in 50 ml of liquid nutrient medium (Sabouraud medium, wort) and incubated at +37°C for 5 days. Methods for rapid identification of C. albicans are widely used. This species of Candida is capable of forming germ tubes and short filaments of pseudomycelium within 2–4 hours at +37°C on blood serum, egg white and other similar media. For the species C.albicans, this phenomenon is typical in 90% of cases. For effective treatment, it is necessary to strive to determine the species of Candida fungi and determine the individual sensitivity of the strain to antimycotic agents; some strains of Candida lusitanlae are resistant to amphotericin, and Candida krusei and Candida glabrata are resistant to fluconazole. Detection of Candida in the blood allows a diagnosis of generalized candidiasis to be made only in combination with appropriate clinical symptoms (repeated detection of Candida in the blood is especially informative). It should be remembered that in 70–80% of patients who actually suffer from generalized candidiasis, fungi cannot be detected by blood culture. The value of serological methods consists mainly in identifying patients with probable invasive mycoses. False-positive results of serological tests are possible with mycocarriage and in healthy people sensitized by fungal antigens; false-negative tests can occur in immunodeficiency. Original procedures for detecting antigens and antibodies of certain metabolites of mushroom cells are proposed; Special diagnostic sets have been created. As an example, Pastorex Candida can be cited - to determine in the reaction of “latex - aglutination” of repeating oligomannous epitopes of antigenic structures expressed on a large number of macromolecules of the fungus. The Platelia Candida set can be used to determine the Candida antigen -mannan, for example, in the blood serum of the patient with microorganism circulation. Using the first set, the threshold for determining antigenic structures is 2.5 ng/ml, using the second in a bunch with the method of determination threshold - 0.5 ng/ml. In the diagnosis of non -invasive intestinal candidiasis, in which there is no tissue biopsy material for mycological examination, the following criteria are proposed as a diagnostic standard: growth of more than 1000 CFU/g Candida SPP. When sowing intestinal contents taken under sterile conditions, in combination with the phenomena of intestinal dyspepsia and positive clinical - laboratory dynamics in the treatment of antimycotic drugs [1]. Unfortunately, the correct fence of intestinal contents for cultural research is technically complicated; The methodology “sowing feces on dysbiosis” in our country cannot serve as a support in assessing the real composition of the intestinal microflora [1.3.5]. In the diagnosis of any form of candiaosis of the digestive organs, it is important to take into account the presence of predisposing risk factors in the patient. The “random” detection of candidiasis should serve as an incentive to the search for such a background factor. It is important to remember that candidiasis can act as an early manifestation of general diseases, accompanied by the development of immunodeficiency. Given the above, it would probably not be entirely correctly formulated by the diagnosis briefly as “candidiasis”, without indicating the background state. The differential diagnosis of invasive intestinal candidiasis (in the detection of macroscopic intestinal changes) should be carried out with chronic inflammatory intestinal diseases, antibiotic -absorbed diarrhea, due to C. Difficile, malignant lesion, ischemic colitis. The non -invasive form of candidiasis should be differentiated with a wide spectrum of enteritis and colitis of other etiology. Indirect evidence in favor of the presence of intestinal candidiasis can be extra -coching systemic manifestations of candidiasis. Treatment should be emphasized once again that only the detection of mushrooms of the genus Candida in a bacteriological analysis of bowel movements (according to the adopted methodology in Russia), regardless of the presence or absence of symptoms of intestinal dyspepsia, cannot serve as an indication for the patient prescribing antimicotic drugs. For the treatment of intestinal candidiasis, it is necessary to prescribe drugs that are non -adsorbed from the intestinal lumen. Today there are a variety of antimycotic agents. Drugs such as amphotericin B, itraconazole, ketoconazole, fluconazole, have a systemic effect, can be used local, orally and intravenously. When prescribed orally, these drugs are almost completely adsorbed from the upper sections of the gastrointestinal tract and do not reach the level of the ileum, where the main population of mushrooms is concentrated. In addition, the use of “systemic” antifungal drugs is often accompanied by side effects, in particular, the development of toxic hepatitis. Almost non -adsorbing antimycotic agents include Levorin, Nystatin and Namycin (Pimafucin). The purpose of Levorin and Nystatin with a fairly high frequency is accompanied by the development of side effects (dyspeptic phenomena, allergies, toxic hepatitis). Pimafucin (Namitzin) is a wide range of a wide -spectrum. It has fungicidal potential. Pimafucin binds sterols of cell membranes, disrupting their integrity and functions, which leads to the death of microorganisms. Most pathogenic yeast mushrooms are sensitive to Natamycin, to the greatest extent - Candida albicans. Pimafucin has higher efficiency compared to nystatin. There were no cases of resistance to Natamycin in clinical practice; With multiple use of this drug, the minimum vast majority of it in relation to C.albicans does not change. Pimafucin in tablets acts only in the lumen of the intestines, practically does not suck from the gastrointestinal tract. When using tablets in the first days of treatment, dyspepsic phenomena are possible - nausea and diarrhea that do not require the cancellation of the drug and are independently resolved during treatment. The only contraindication to the purpose of Pimafucin is increased sensitivity to the components of the drug. Pimafucin can be prescribed during periods of pregnancy and lactation, as well as newborn children. For the treatment of intestinal candidiasis, it is necessary to conduct a course of treatment with non -disinfected antifungal drugs for 7-10 days. Nystatin is prescribed 250,000 units 6–8 times a day (daily dose - up to 3 million units) for 14 days. It is prescribed 100 mg (1 tablet) 2–4 times a day for 7-10 days [1]. Based on the above provisions and clinical experience, it is necessary to admit that the tactics for the treatment of candidiasis of mucous membranes of extracurricular localization only by local antifungal agents or systemic drugs, absorbed in the gastrointestinal tract, are inherently erroneous. Since the intestines are the source of lymph and gematogenic distribution of virulent Candida virulent strains, the intestines are, without the suppression of mushroom growth in its lumen, antifungal therapy is ineffective or only a short -term inconsistent effect is observed. With systemic candidiasis, the dose of nystatin can be increased to 4-6 million units/day, at the same time a local active antifungal agent is prescribed. For systemic candidiasis, Pimafucin is used in the same dose while the appointment of a local acting antifungal agent. In severe cases, drugs with systemic effects are added to complex therapy [1.5]. Timely recognition and therapy of intestinal candidiasis, such as the prevention of systemic and generalized candidiasis, are especially important, in patients with risk groups, which include primarily patients receiving antitumor radial and/or chemicals, anti -tuberculosis drugs, patients preparing for planned abdominal organs. In these situations, it is most preferable to prescribe non -subclarb drugs, since their long -term and repeated intake does not significantly affect the pharmacodynamics of other drugs. The main criterion for the effectiveness of therapy is not to obtain a negative result of sowing on mushrooms, but primarily the disappearance of the main manifestations of the disease, the normalization of the number of fungi according to mycological examination (if possible adequate sowing of the intestinal contents). To achieve the effect, you often have to resort to repeated treatment courses [1]. This article set the goal of expanding the presentation of practical doctors about the flexibility of the interaction of the human body with symbionic microflora, about the pathogenic potential, which is possessed by the harmless intestinal commensal - Candida. The internalists and specialists in medical mycology are currently faced with a distinct determination of the prevalence and clinical significance of fungal infections in clinical practice, the development of an algorithm for diagnosis and indications for the treatment of intestinal candidiasis. Literature 1. Zlatkina A.R., Isakov V.A., Ivanikov I.O. Candidiasis of the intestine as a new problem of gastroenterology. // Russian journal of gastroenterology, hepatology, coloproctology. - 2001. - No. 6. - S.33–38. 2. Danna Pl, Urban C., Bellin E., Rahal JJ Role of Candida in Pathogenesis of Antibiotic - Ssociated Diarrhoea in Elderly Patients./ - - 1991.-vol.337.–p.511–514. 3. Presscott RJ, Harris M., Banerjee SS Fungal Infections of Small and Large Intestine.//j.clin.path. --Vol.45. - P.806–811. 4. Redmond HP, Shou J., Kelly CJ et al. Protein - Calorie Malnutrition Impairs Host Defense Against Candida albicans.//j.Surg.Res.-VOL.50. - P.552–559. 5. The Merck Manual. Sixteenth Edition. Copyright (C) 1992 by Merck & Co., Inc.

Authorized Products

A diet for thrush requires the inclusion in the diet of foods rich in protein and plant fiber.
These include:

1. Meat products: lean beef, turkey, chicken, rabbit.
2. Seafood: lean fish, kelp, mussels, oysters.
3. Vegetables and herbs: dill, parsley, onions, broccoli, cauliflower, cucumbers, green peas, carrots, sorrel, celery, spinach, eggplant, radish, zucchini, etc.
4. Berries: viburnum, currants, grapefruit, lingonberries, currants, rowan.
5. Fruits: lemons, oranges, apples, pomegranates.
6. Seasonings: garlic, black pepper, oregano, bay leaf, cloves, cinnamon.
7. Dairy products: low-fat cottage cheese, biokefir, fermented baked milk, natural yogurt.
8. Cereals and porridges: buckwheat, brown and brown rice, oatmeal.
9. Oils: sunflower, olive, corn, butter.
10. Eggs: quail and chicken.
11. Drinks: tomato and carrot juices, lingonberry and pumpkin fruit drinks, mineral water.

For candidiasis, meat products are allowed.

To compensate for a possible lack of protein and nutrients, it is advisable to introduce seeds and nuts into the diet.

You can consume whole wheat pasta and whole grain bread in limited quantities.

During pregnancy

During pregnancy, a woman experiences changes in her body and pronounced hormonal changes.
This creates conditions for the activation of pathogenic fungi that cause candidiasis. If symptoms of this disease begin to appear after conception, you should consult a doctor who can select medications that are safe for the fetus. Women planning to become pregnant are advised to follow a special diet in advance. The diet is selected so that it satisfies the body’s needs for vitamins, minerals and other substances, but does not contribute to the growth of pathogenic fungi.

During this period, you need to exclude foods containing easily digestible carbohydrates, incl. confectionery, honey, sugar, etc. You should stop eating foods that have been prepared with yeast. In addition, you should not consume milk or cream during treatment. It is also important to exclude cereals and vegetables with a high starch content, as well as sweet fruits and drinks made from them.

The basis of the diet should be the following products:

• lean varieties of fish and meat;
• eggs;
• cabbage;
• salad;
• cucumbers;
• turnip;
• zucchini;
• carrot;
• seaweed;
• sprouted grains;
• eggplant;
• tomatoes;
• legumes;
• low-fat sour cream;
• tofu cheese;
• kefir;
• pickled vegetables.

Pregnant women need to follow a diet.

In addition, it is recommended to use compotes and decoctions of cranberries, currants and lingonberries. Be sure to drink carrot, pumpkin and citrus juices.

Switching to a thrush diet

After the cleansing stage is completed, you can move on to an antifungal diet, which not only prevents the development of candidiasis, but also helps the body get rid of this disease forever. The diet consists of several stages.

Refusal of problematic products

First of all, you need to continue to remove foods from your diet that are literally a breeding ground for candida and stimulate its growth in the body. Common pathogens include sugar, white flour, yeast and alcohol. These foods are thought to promote the growth of candida. If you avoid sugar and white flour, you can easily avoid most other processed foods, which tend to be higher in calories, unhealthy ingredients, and lower in nutrients. Avoiding sugar in all its different forms is truly the key to a candida diet in women. Yeast cells need sugar to build cell walls, reproduce, and transform into a more dangerous fungal form. This is why a low sugar diet is such a necessary part of treatment. The diet should focus on vegetables, high-quality protein foods, and gluten-free grains such as brown rice and millet. Avoiding fruits during this time is also usually recommended because although fruits are very healthy, they turn into sugar in the body. As for vegetables, you should also avoid these sweet, starchy vegetables: potatoes, carrots, sweet potatoes, beets, peas. These vegetables are prohibited in a strict anti-candida diet due to their high carbohydrate content, but they are certainly rich in nutrients and can be introduced into the diet later.

Strengthening the immune system

There are 10 special foods to include in your daily list to boost your immune system and inhibit yeast growth, including apple cider vinegar, sauerkraut and other fermented vegetables, green vegetables and green drinks, coconut oil, honey, garlic, flaxseed and oil, natural cranberry juice, fermented milk products, as well as spices such as turmeric and cinnamon. On the subject: Why you can’t eat after 6 pm To achieve results with this diet for candidiasis of the esophagus and other organs, you need from several weeks to several months. This depends on a few key elements:

• How strictly is the diet followed?
• Consumption of probiotics and antifungals.
• Patient's immunity.
• Tendency to such diseases and lifestyle.

Of course, you shouldn’t forget about personal hygiene, the right choice of antifungal agents, consultation with your doctor, smoking and other bad habits that also affect the effectiveness of the diet.

Reintroduction of Prohibited Products

As soon as the first results are visible and the thrush begins to recede, many break down and return to their previous diet and lifestyle. Naturally, thrush reappears on the horizon. To prevent this from happening, you need to gradually introduce some foods back into your diet. Low-sugar fruits like green apples are a great example of smart choices. If reintroduced foods do not aggravate candidiasis symptoms, you can continue to check the list further. You can introduce approximately one product every 3-4 days in order to closely monitor the body’s reaction and, if anything, immediately abandon the problematic food. There is a basic list of products that are recommended to be included in your daily diet for the prevention and treatment of candidiasis:

1. Apple vinegar. The acid and enzymes in apple cider vinegar help rid the body of excess yeast.
2. Sauerkraut. Fermented foods, such as sauerkraut, contain microflora that help protect the intestines. Regular consumption can help improve the immune system, making the body less susceptible to candida.

1. Green vegetables and green drinks. Leafy green vegetables help combat the acidic nature of yeast overgrowth. Greens are sugar-free but contain high levels of magnesium to naturally detoxify the body, vitamin C to strengthen the immune system, chlorophyll to cleanse the body, B vitamins to energize, and iron for overall support.
2. Coconut oil. It has antimicrobial properties, and the combination of lauric and caprylic acids contained in coconut oil kills harmful microflora when consumed and applied topically.
3. Stevia. Stevia is an ideal choice for those on a diet, as it is an excellent substitute for sugar, sweetening foods. Stevia is not only antifungal, anti-inflammatory and antibiotic, but also helps balance the pancreas.
4. Garlic. Contains a large number of sulfur-containing compounds that have extremely strong broad-spectrum antifungal properties. Raw garlic is especially useful for fighting thrush.

1. Ground flax seeds. The polyphenols found in flax seeds support the growth of probiotics in the gut and may also help eliminate candida in the body.
2. Unsweetened cranberry juice. Cranberry juice without added sugar helps adjust urine pH levels, helping prevent fungal overgrowth.
3. Dairy products. Cultured dairy products, preferably kefir, can effectively destroy candida in the body and promote the growth of healthy microflora.
4. Spices such as turmeric and cinnamon. A diet for the treatment of candidiasis is impossible without spices. Turmeric contains an active ingredient called curcumin, which has been shown to completely inhibit the growth of Candida albicans (as well as many other fungal strains). Cinnamon may treat thrush because studies have shown that people who consume cinnamon tend to suffer from this condition much less frequently.
5. Cooked vegetables. Non-starchy cooked vegetables such as broccoli, cauliflower and asparagus contain valuable nutrients that fight thrush.

6. Meat of natural origin. Protein plays a key role in an anti-thrush diet. If you obtain protein from factory-raised meat, you may not fully know what auxiliary products and substances were used and how they will affect the development of thrush. That's why it's important to eat only organic meat.
7. Bone broth. Beneficial for many aspects of health. This is one of the best sources of candida-killing substances due to the positive effects of the broth on gut health.

List of foods that need to be excluded from the diet

It is extremely important to exclude from the diet all foods that can activate the growth of fungi.
When preparing dishes, salt can be replaced with a small amount of vinegar or lemon juice. The list of foods not recommended for consumption with thrush includes:

• rye;
• barley;
• wheat;
• bananas;
• raisin;
• dates;
• grape;
• mango;
• peanut;
• cheese;
• full fat milk;
• soy sauce;
• sugar;
• confectionery;
• ketchup;
• fatty meats and fish;
• sausages;
• honey;
• cream, etc.

Bananas should be excluded from the diet.

You should avoid drinking sugary carbonated drinks, concentrated fruit juices and alcohol.

In addition, it is necessary to exclude all products containing artificial sweeteners, nitrates and sulfates.

General rules of nutrition in the presence of illness

The diet should be followed not only throughout the active period of the disease, but also for at least one and a half months after the symptoms have resolved. The calorie content of the diet should be 1900-2200 kcal per day. Be sure to drink at least 1.5 liters of water. In this case, the share of plant foods should reach 60%. If possible, dishes should be prepared without adding salt and spices. Thermal processing of products is carried out by boiling, stewing, steaming, etc.

For thrush in women

In women, thrush occurs with severe symptoms and often becomes recurrent, so it is necessary to limit as much as possible the consumption of products that provoke the rapid proliferation of pathogenic microflora.

It is necessary to limit the consumption of foods that provoke the proliferation of microflora.
It is necessary to introduce into the diet foods that have antifungal and antibacterial effects. Natural yoghurts, low-starch vegetables, seafood, herbs and garlic are extremely healthy. In addition, it is recommended to drink teas made from alfalfa, chamomile, clover, etc.

For candidiasis in men

In men, candidiasis with pronounced manifestations is rare.
This disease requires a diet that enhances immunity. Men are advised to increase their intake of protein-rich foods and, if possible, avoid foods containing large amounts of simple carbohydrates. In this case, a special diet should be followed for at least 4-12 months to reduce the risk of relapse of the disease. During the period of therapy it is necessary to avoid alcoholic beverages, sauces and hot spices. Preference should be given to easily digestible dishes.

Prebiotics of natural origin

These components also help to cope with fungus in the oral cavity and increase the effectiveness of treatment. Prebiotics, according to experts, are complex polysaccharides that are not digested by enzymes.

As a result, they become excellent food for intestinal microflora and displace pathogenic microorganisms. Fructose-oligosaccharide is rightfully recognized as one of the most famous polysaccharides. Its maximum content was found in:

• Asparagus;
• Bananas;
• Onions and garlic;
• Jerusalem artichoke.

Introducing the above ingredients into your daily diet will reduce the risk of relapse and speed up the recovery process.

Symptoms of thrush in the esophagus and stomach

Fungal infections of the esophagus and stomach are rare.
In most cases, this disease occurs with a severe decrease in immunity. Characteristic manifestations of candidiasis of the esophagus and stomach include:

• heartburn;
• discomfort when swallowing;
• sore throat;
• vomiting with mucus discharge;
• decreased appetite;
• stool disorders;
• feeling of a lump in the throat.

Heartburn is manifested by discomfort or a burning sensation in the chest.
This type of thrush is characterized by a long latent period of the disease. This results in a delay in starting treatment. The examination reveals redness and swelling of the mucous membrane of the esophagus and stomach, as well as a characteristic white coating.

In the later stages of development of the pathology, deep layers of tissue are affected, which leads to the formation of ulcers and scars.

Features of lower back pain in women

Back pain is almost never the only complaint - in many cases the patient notes the presence of other symptoms that bother him. In women, it can be combined with pain in the lower abdomen and urination problems. In such a situation, a thoughtful doctor will refer the patient, first of all, for an examination to a gynecologist to rule out pathology of the “female part” or pregnancy.

For women's diseases

If a woman’s lower back hurts, the common causes of this condition are as follows:

• endometriosis (accompanied by aching pain in the lower back and nagging pain in the abdomen, one- or two-sided, abundant and prolonged, with clots, menstruation, irregular cycles);
• uterine fibroids (with this disease, the uterus, as during pregnancy, gradually increases in size, squeezing the internal organs and tissues surrounding the uterus, which causes lower back pain);
• uterine polyposis (occurs with heavy menstruation, spotting vaginal discharge after intercourse; women also report pain in the lower abdomen or behind, in the sacral spine, tailbone);
• prolapse, prolapse of the uterus, vagina (with sudden movements, bending of the torso, patients experience aching pain in the lower abdomen, often radiating to the lower back, legs; urination disorders are also noted (frequent urge, pain) and increased frequency of bowel movements);
• polycystic ovary syndrome (the most characteristic symptom is pain in the iliac region from the side of the affected appendage; in some cases it radiates to the lower back, intensifies with physical activity, bending the body, and also in the evening).

During pregnancy

A feeling of discomfort, heaviness, nagging, low-intensity pain in the lower abdomen and lower back in the absence of other warning signs for the woman and the doctor is a normal option that does not require concern or treatment. These symptoms arise due to the pressure of the growing uterus on the surrounding tissues, a shift in the center of gravity due to the increasing size of the abdomen, and also due to the active production of progesterone by the body of the expectant mother, which leads to some weakness of the muscles and ligaments. They are often accompanied by nausea and vomiting.

If the pain in the lower back is intense, accompanied by severe cramping pain in the abdomen, this is a sign of a threat of miscarriage, requiring urgent consultation with a doctor.

After childbirth

Any woman knows that the lower back can get sore after childbirth. This pain is caused by weakening of the muscles of the anterior abdominal wall during pregnancy, and less often by injuries to elements of the mother’s musculoskeletal system during childbirth.

Diet for intestinal candidiasis

You need to consume as many fermented milk products as possible, which contain a lot of lactobacilli.
This will help restore normal microflora. The diet should include foods that are not capable of damaging the already inflamed mucous membrane. Recommended ones include:

• all types of cabbage;
• natural yogurt;
• biokefir;
• cottage cheese;
• bird;
• lean meat;
• seafood;
• whole oatmeal;
• brown rice porridge;
• buckwheat;
• bananas;
• artichokes;
• leek;
• Jerusalem artichoke;
• apples;
• beans;
• natural juices from cranberries and wheat germ, etc.

For intestinal candidiasis, cabbage is recommended.
For intestinal candidiasis, drinking plenty of fluids is recommended. You can drink rosehip decoction, chamomile tea, and non-acidic freshly squeezed juices. In this case, you need to give up yeast bread and pastries, sugar, honey, confectionery and other foods rich in simple carbohydrates.

Nutrition for oral candidiasis

For thrush of the oral mucosa, it is necessary to adhere to the same recommendations regarding nutrition as for intestinal damage. The diet should be gentle and at the same time help improve immunity.

Cooking food has a great influence on its final properties.

Cooking Features

To quickly eliminate the symptoms of oral candidiasis, you need to eat dishes prepared using gentle methods, incl. by cooking, steaming and stewing. Porridges should be made slimy. When preparing dishes, do not use salt or hot spices.

Recommended Products

If the oral mucosa is damaged, vegetables and fruits with low acid content can be introduced into the diet.
Recommended foods and dishes include:

• cauliflower;
• lean varieties of fish and meat;
• bird;
• dairy products;
• slimy porridge;
• compotes;
• boiled fruits, etc.

If the oral mucosa is damaged, include cauliflower in your diet.

It is permissible to consume dried unleavened bread in limited quantities.

Dish recipes

There are many delicious dishes that can be prepared from products recommended for candidiasis. You should consider recipes for simple first and second courses.

Zucchini pancakes

To make zucchini pancakes you will need the following ingredients:

• young zucchini - 3 pcs.;
• onion - 1 pc.;
• carrots - 1 pc.;
• eggs - 2-3 pcs.;
• greens - a bunch.

Zucchini pancakes are a vegetable dish made from zucchini.
The zucchini is peeled and ground. The pulp is slightly salted and after 15 minutes the released juice is eliminated. Grate the carrots and cut the onion into small cubes. All components are mixed.

Chopped herbs and eggs are added to them. Cutlets are formed from the resulting mixture and fried in olive oil.

Omelette with spinach

Another delicious dish that can be consumed when treating candidiasis is spinach omelette.
To prepare it you will need:

• eggs - 2-3 pcs.;
• milk - 2 tbsp;
• spinach - 80 g.

Spinach omelette is a delicious and quick breakfast option.
First you need to wash and chop the greens. Then fry the spinach for 1-2 minutes in a hot frying pan with olive oil. Combine eggs with milk and beat lightly. After this, pour the mixture into the pan and close the lid. The finished dish can be sprinkled with fresh herbs.

Beet salad

This salad can be prepared quickly if you already have cooked beets. This vegetable must be grated on a coarse grater, and a crushed clove of garlic must be added to the mixture. It is recommended to dress the salad with low-fat sour cream.

Beetroot salad is tasty, healthy and safe for your figure.

Green borscht with sorrel

Among the dishes that are acceptable to eat for candidiasis, such a tasty dish as green borscht stands out.
To prepare it you will need:

• broth - 2.5 l;
• onion - 1 pc.;
• potatoes - 3-4 pcs.;
• carrots - 1 pc.;
• spinach - 1 bunch;
• sorrel - 1 bunch;
• tomato paste - 1-2 tbsp;
• olive oil - 30 g.

Green borscht with sorrel is the first dish that is best prepared in the summer.
The potatoes are peeled and cut into large cubes. The carrots should be grated, the onions should be chopped into small pieces, and the greens should be cut into thin strips.

Potatoes are added to the boiling salted broth. Carrots and onions are lightly fried in vegetable oil. Tomato paste is then added to these vegetables. After 3-5 minutes of simmering, remove the roast from the heat. When the potatoes are almost cooked, frying and chopped herbs are added to the broth. After 10 minutes the dish is ready.

Steam omelette

Steamed omelet is another dish that can be quickly prepared at home.

Steam omelet is a nutritious egg dish.

To do this, break 2 eggs into a container and add 2-3 tbsp to them. milk. Then place the container with the egg mixture in a colander, and place it in the pan so that it does not touch the water poured to the bottom. The omelette should be cooked for 10-15 minutes.